He was "cured" by a bone marrow transplant from a donor carrying the Delta-32 mutation, causing him to lack the CCR5 receptor, a cell-surface receptor present on antigen presenting cells of the immune system. This is not the only receptor that HIV can bind to and infect cells through, though. The CXCL4 receptor on activated T-cells is another target of the virus, and unless the donor is magically missing that one too, this guy still has HIV, and could spread it. He might more easily be able to fight the virus, especially in combination with typical HAART treatment (and considering his unique case, it's undoubted he would be receiving the therapy), but he is most definitely still HIV+, and the virus is still reproducing in his immune system, albeit without the help of using his APC's as a vector anymore.
It's not a cure, but it's a start."
A start is a damned good thing, at any rate, I think we can all agree.
no subject
(no subject)
no subject
"Okay, science time!
He was "cured" by a bone marrow transplant from a donor carrying the Delta-32 mutation, causing him to lack the CCR5 receptor, a cell-surface receptor present on antigen presenting cells of the immune system. This is not the only receptor that HIV can bind to and infect cells through, though. The CXCL4 receptor on activated T-cells is another target of the virus, and unless the donor is magically missing that one too, this guy still has HIV, and could spread it. He might more easily be able to fight the virus, especially in combination with typical HAART treatment (and considering his unique case, it's undoubted he would be receiving the therapy), but he is most definitely still HIV+, and the virus is still reproducing in his immune system, albeit without the help of using his APC's as a vector anymore.
It's not a cure, but it's a start."
A start is a damned good thing, at any rate, I think we can all agree.
Nope.
no subject
Poor nature. It has yet to win.
( This is brilliant. ♥ )
(no subject)
(no subject)
(no subject)